Meanwhile, the amygdalae primarily regulate the intensity of an emotional reaction to a traumatic event, and hence affect the arousal of other brain structures. Too rapid or too long secretion of stress hormones may lead to an excessive arousal of the hippocampus, which may then lead to hippocampus damage. As a result of the exposure to stress factors, stress hormones are secreted, which leads to an arousal of the hippocampus. The hip-pocampus is a very flexible structure, yet highly vulnerable. In the case of an excessive arousal, its functions become deregulated, which results in the inability to register the received stimuli. The prefrontal cortex is a structure, which integrates internal and external experience. Research on this phenomenon indicated that the main biological factors that trigger dissociative amnesia are an excessive arousal of the prefrontal cortex, hippocampus and amygdalae. Although the classification criteria of dissociative amnesia are based solely on the clinical image and do not refer to biological mechanisms, recent neurobiolo-gical studies using applied modern brain imaging techniques suggest that biological mechanisms play a crucial role in this disorder. Dissociative amnesia is defined as the retrograde memory loss in the absence of detectable structural brain damage caused by disassociation.

A hint of amnesia